IRIS - Res&Arch Institutional Research Information System - Research & Archive

IMBRICI, Paola
 Distribuzione geografica
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Continente #
NA - Nord America 880
EU - Europa 736
AS - Asia 224
SA - Sud America 2
Totale 1.842
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Nazione #
US - Stati Uniti d'America 877
UA - Ucraina 198
IE - Irlanda 195
SG - Singapore 93
FR - Francia 79
SE - Svezia 66
HK - Hong Kong 58
DE - Germania 52
IT - Italia 41
FI - Finlandia 37
CN - Cina 28
RU - Federazione Russa 26
VN - Vietnam 26
GB - Regno Unito 16
BE - Belgio 9
KR - Corea 9
CH - Svizzera 7
TR - Turchia 7
AT - Austria 3
BR - Brasile 2
CA - Canada 2
GR - Grecia 2
NL - Olanda 2
CZ - Repubblica Ceca 1
IL - Israele 1
LB - Libano 1
MX - Messico 1
PL - Polonia 1
RO - Romania 1
UZ - Uzbekistan 1
Totale 1.842
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Città #
Dublin 194
Chandler 169
Jacksonville 108
San Mateo 73
Singapore 67
Ann Arbor 60
Hong Kong 58
Boardman 54
Santa Clara 41
Medford 37
Princeton 36
Wilmington 30
Andover 26
Dong Ket 26
Des Moines 18
Saint Petersburg 14
Ashburn 11
Altamura 10
Brussels 9
Lawrence 9
Los Angeles 8
Norwalk 8
Perugia 8
Beijing 6
Izmir 6
Seoul 6
Woodbridge 6
Auburn Hills 5
Munich 5
Dallas 4
Dearborn 4
Helsinki 3
Shanghai 3
Boscoreale 2
Chemnitz 2
Edinburgh 2
Fairfield 2
Frankfurt Am Main 2
Redwood City 2
Rome 2
San Paolo di Civitate 2
Changsha 1
Costa Mesa 1
Den Haag 1
Deruta 1
Gatineau 1
Istanbul 1
Kiev 1
Kilkenny 1
Lafayette 1
Leverano 1
Magione 1
Mannheim 1
Moscow 1
Narni 1
Oakville 1
Parma 1
Porto Alegre 1
San Jose 1
Shijiazhuang 1
Siena 1
São Paulo 1
Tel Aviv 1
Timisoara 1
Vercelli 1
Totale 1.162
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Nome #
Voltage-gated calcium channels modulate synaptic transmission at vestibular neurons. 90
Episodic Ataxias as Ion Channel Diseases. 81
Modulation of hKv 1.1 and hKv 1.2 voltage gating and C-type inactivation by 5-HT2C receptors. 77
The Episodic Ataxia Type 1 Mutation F184C Alters the Zn2+ Modulation of the Human Kv1.4-Kv1.1/Kvbeta1 Channel 68
All atom molecular dynamics simulation of the K+ Channel Chimera Kv1.2/2.1 67
Effects of Episodic Ataxia-Associated Mutations on hKv1.4-1.1/Kvbeta1 channels 66
The emerging role of the inwardly rectifying K+channels in autism spectrum disorders andepilepsy 65
Episodic Ataxia Type 1 Mutations Affect Fast Inactivation of K+ Channels by a Reduction in Either Subunit Surface Expression or Affinity for Inactivation Domain 63
Autism with Seizures and Intellectual Disability: Possible Causative Role of Gain-of-Function of the Inwardly-Rectifying K+ Channel Kir4.1. 57
Contributions of the central hydrophobic residue in the PXP motif of Voltage-Dependent K+ Channels to S6 flexibility and Gating Properties. 56
Identification and functional characterization of a novel mutation in the KCNA1 gene of a Sicilian family affected by episodic ataxia type 1 55
Functional Properties of Voltage-Gated Potassium Channels Probed with Methanethiosulfonate Reagents. 55
Mutations in KCNA1 gene associated with episodic ataxia type-1 sindrome impair heteromeric voltage-gated K+ channel function. 54
Gating properties of human heteromeric voltage-gated potassium channels and effects of episodic ataxia type-1 mutations. 54
Episodic Ataxia Type 1 Mutation F184C Alters Zn2+-Induced Modulation of the Human Potassium Channel Kv1.4-Kv1.1/Kvbeta1.1 53
Trace amines depress D2-autoreceptor-mediated responses on midbrain dopaminergic cells. 51
Episodic ataxia type 1 mutations cause loss-of-function impairments of heteromeric channels formed by the Kv1.4 and Kv1.1 subunits. 51
CIC-1 chloride channels: State-of-the-art research and future challenges 51
The Episodic Ataxia Type 1 Mutation F184C Alters the Zn2+ Modulation of the Human Kv1.4-Kv1.1/Kvbeta1 Channel 50
Mutations in KCNA1 affect stoichiometry and fast inactivation of heteromeric K+ channels 50
Role of inwardly-rectifying potassium channels Kir5.1 in learning and memory processes in a mouse knock-out model. 49
EA-1 mutations alter the fast inactivation properties of Kv1.1 channels conferred by Kv1.4 and Kvbeta1.1 subunits 46
Identification of a New de Novo Mutation Underlying Regressive Episodic Ataxia Type I 46
Functional Characterization of an Episodic Ataxia Type-1 Mutation Occurring in the S1 Segment of hKv1.1 Channels. 45
Modulation of delayed rectifier K+ channels by 5HT2c receptors. 45
Episodic Ataxia Type 1 Mutations in the KCNA1 Gene Impair the Fast Inactivation Properties of the Human K+ Channels Kv1.4-1.1/Kvbeta1.1 and Kv1.4-1.1/Kvbeta1.2. 44
Differential pH-sensitivity of Kir4.1 and Kir4.2 and modulation by heteropolymerisation with Kir5.1 43
Genetic inactivation of Kcnj16 identifies Kir5.1 as an important determinant of neuronal PCO2/pH sensitivity 42
Role of the S1 segment in the voltage-dependent gating of Kv1.1 channels revealed by naturally occurring pathogenic mutations. 41
Copertina della rivista The Journal of Physiology 41
Electromechanical coupling of the Kv1.1 voltage-gated K+ channel is fine-tuned by the simplest amino acid residue in the S4-S5 linker 41
The Kir5.1 Potassium Channel is an Important Determinant of Neuronal PCO2/pH Sensitivity 40
A Novel KCNA1 Mutation Identified in an Italian Family Affected by Episodic Ataxia Type 1 39
The neurobiology of episodic ataxia type 1: a Shaker-like K+ channel disorder. 35
Gain-of-Function of the Inwardly-Rectifying K+ Channel Kir4.1 Contributes to Autism with Seizures and Intellectual Disability. 34
A large cohort of myotonia congenita probands: novel mutations and a high-frequency mutation region in exons 4 and 5 of the CLCN1 gene. 29
An activator of voltage-gated K + channels Kv1.1 as a therapeutic candidate for episodic ataxia type 1 14
Kcnj16 (Kir5.1) Gene Ablation Causes Subfertility and Increases the Prevalence of Morphologically Abnormal Spermatozoa 8
Totale 1.896
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Categoria #
all - tutte 7.969
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 7.969
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2019/2020164 0 0 0 0 31 26 34 8 29 16 4 16
2020/2021332 3 29 14 33 100 12 29 2 37 10 34 29
2021/2022264 8 44 9 16 2 0 5 106 3 9 17 45
2022/2023561 41 33 22 34 61 66 0 31 246 2 19 6
2023/2024192 17 23 11 1 4 0 36 1 24 11 35 29
2024/2025174 7 54 10 29 74 0 0 0 0 0 0 0
Totale 1.896