IMBRICI, Paola
 Distribuzione geografica
Continente #
NA - Nord America 778
EU - Europa 728
AS - Asia 169
SA - Sud America 1
Totale 1.676
Nazione #
US - Stati Uniti d'America 775
UA - Ucraina 198
IE - Irlanda 195
FR - Francia 79
SE - Svezia 66
HK - Hong Kong 58
DE - Germania 48
SG - Singapore 43
IT - Italia 39
FI - Finlandia 36
RU - Federazione Russa 26
VN - Vietnam 26
CN - Cina 23
GB - Regno Unito 16
BE - Belgio 9
KR - Corea 9
CH - Svizzera 7
TR - Turchia 7
AT - Austria 2
CA - Canada 2
GR - Grecia 2
NL - Olanda 2
BR - Brasile 1
CZ - Repubblica Ceca 1
IL - Israele 1
LB - Libano 1
MX - Messico 1
PL - Polonia 1
RO - Romania 1
UZ - Uzbekistan 1
Totale 1.676
Città #
Dublin 194
Chandler 169
Jacksonville 108
San Mateo 73
Ann Arbor 60
Hong Kong 58
Medford 37
Princeton 36
Wilmington 30
Andover 26
Dong Ket 26
Singapore 24
Des Moines 18
Saint Petersburg 14
Altamura 10
Boardman 9
Brussels 9
Lawrence 9
Norwalk 8
Perugia 8
Los Angeles 7
Beijing 6
Izmir 6
Seoul 6
Woodbridge 6
Auburn Hills 5
Ashburn 4
Dearborn 4
Shanghai 3
Chemnitz 2
Edinburgh 2
Fairfield 2
Frankfurt Am Main 2
Helsinki 2
Munich 2
Redwood City 2
Rome 2
San Paolo di Civitate 2
Costa Mesa 1
Den Haag 1
Deruta 1
Gatineau 1
Istanbul 1
Kiev 1
Kilkenny 1
Lafayette 1
Leverano 1
Magione 1
Mannheim 1
Moscow 1
Narni 1
Oakville 1
Parma 1
Porto Alegre 1
San Jose 1
Siena 1
Tel Aviv 1
Timisoara 1
Vercelli 1
Totale 1.012
Nome #
Voltage-gated calcium channels modulate synaptic transmission at vestibular neurons. 82
Episodic Ataxias as Ion Channel Diseases. 78
Modulation of hKv 1.1 and hKv 1.2 voltage gating and C-type inactivation by 5-HT2C receptors. 73
The Episodic Ataxia Type 1 Mutation F184C Alters the Zn2+ Modulation of the Human Kv1.4-Kv1.1/Kvbeta1 Channel 63
Effects of Episodic Ataxia-Associated Mutations on hKv1.4-1.1/Kvbeta1 channels 62
All atom molecular dynamics simulation of the K+ Channel Chimera Kv1.2/2.1 62
The emerging role of the inwardly rectifying K+channels in autism spectrum disorders andepilepsy 60
Episodic Ataxia Type 1 Mutations Affect Fast Inactivation of K+ Channels by a Reduction in Either Subunit Surface Expression or Affinity for Inactivation Domain 55
Autism with Seizures and Intellectual Disability: Possible Causative Role of Gain-of-Function of the Inwardly-Rectifying K+ Channel Kir4.1. 54
Contributions of the central hydrophobic residue in the PXP motif of Voltage-Dependent K+ Channels to S6 flexibility and Gating Properties. 53
Functional Properties of Voltage-Gated Potassium Channels Probed with Methanethiosulfonate Reagents. 52
Gating properties of human heteromeric voltage-gated potassium channels and effects of episodic ataxia type-1 mutations. 51
Identification and functional characterization of a novel mutation in the KCNA1 gene of a Sicilian family affected by episodic ataxia type 1 50
Mutations in KCNA1 gene associated with episodic ataxia type-1 sindrome impair heteromeric voltage-gated K+ channel function. 50
Episodic Ataxia Type 1 Mutation F184C Alters Zn2+-Induced Modulation of the Human Potassium Channel Kv1.4-Kv1.1/Kvbeta1.1 49
Trace amines depress D2-autoreceptor-mediated responses on midbrain dopaminergic cells. 47
CIC-1 chloride channels: State-of-the-art research and future challenges 47
The Episodic Ataxia Type 1 Mutation F184C Alters the Zn2+ Modulation of the Human Kv1.4-Kv1.1/Kvbeta1 Channel 46
Episodic ataxia type 1 mutations cause loss-of-function impairments of heteromeric channels formed by the Kv1.4 and Kv1.1 subunits. 46
Role of inwardly-rectifying potassium channels Kir5.1 in learning and memory processes in a mouse knock-out model. 43
Functional Characterization of an Episodic Ataxia Type-1 Mutation Occurring in the S1 Segment of hKv1.1 Channels. 42
Episodic Ataxia Type 1 Mutations in the KCNA1 Gene Impair the Fast Inactivation Properties of the Human K+ Channels Kv1.4-1.1/Kvbeta1.1 and Kv1.4-1.1/Kvbeta1.2. 41
EA-1 mutations alter the fast inactivation properties of Kv1.1 channels conferred by Kv1.4 and Kvbeta1.1 subunits 41
Modulation of delayed rectifier K+ channels by 5HT2c receptors. 41
Identification of a New de Novo Mutation Underlying Regressive Episodic Ataxia Type I 41
Mutations in KCNA1 affect stoichiometry and fast inactivation of heteromeric K+ channels 40
Differential pH-sensitivity of Kir4.1 and Kir4.2 and modulation by heteropolymerisation with Kir5.1 40
Genetic inactivation of Kcnj16 identifies Kir5.1 as an important determinant of neuronal PCO2/pH sensitivity 39
Copertina della rivista The Journal of Physiology 38
The Kir5.1 Potassium Channel is an Important Determinant of Neuronal PCO2/pH Sensitivity 37
Role of the S1 segment in the voltage-dependent gating of Kv1.1 channels revealed by naturally occurring pathogenic mutations. 37
A Novel KCNA1 Mutation Identified in an Italian Family Affected by Episodic Ataxia Type 1 36
Electromechanical coupling of the Kv1.1 voltage-gated K+ channel is fine-tuned by the simplest amino acid residue in the S4-S5 linker 36
The neurobiology of episodic ataxia type 1: a Shaker-like K+ channel disorder. 31
Gain-of-Function of the Inwardly-Rectifying K+ Channel Kir4.1 Contributes to Autism with Seizures and Intellectual Disability. 30
A large cohort of myotonia congenita probands: novel mutations and a high-frequency mutation region in exons 4 and 5 of the CLCN1 gene. 26
An activator of voltage-gated K + channels Kv1.1 as a therapeutic candidate for episodic ataxia type 1 9
Totale 1.728
Categoria #
all - tutte 6.479
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 6.479


Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2019/2020226 28 3 30 1 31 26 34 8 29 16 4 16
2020/2021332 3 29 14 33 100 12 29 2 37 10 34 29
2021/2022264 8 44 9 16 2 0 5 106 3 9 17 45
2022/2023561 41 33 22 34 61 66 0 31 246 2 19 6
2023/2024192 17 23 11 1 4 0 36 1 24 11 35 29
2024/20256 6 0 0 0 0 0 0 0 0 0 0 0
Totale 1.728