IRIS - Res&Arch Institutional Research Information System - Research & Archive

RIUZZI, Francesca
 Distribuzione geografica
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Continente #
NA - Nord America 2.774
EU - Europa 1.981
AS - Asia 1.057
SA - Sud America 35
AF - Africa 9
Continente sconosciuto - Info sul continente non disponibili 4
OC - Oceania 3
Totale 5.863
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Nazione #
US - Stati Uniti d'America 2.762
UA - Ucraina 463
IE - Irlanda 434
SG - Singapore 425
IT - Italia 362
HK - Hong Kong 241
SE - Svezia 165
CN - Cina 153
FI - Finlandia 102
DE - Germania 100
VN - Vietnam 96
RU - Federazione Russa 75
GB - Regno Unito 66
KR - Corea 53
RO - Romania 50
AT - Austria 35
BR - Brasile 30
FR - Francia 24
NL - Olanda 21
CH - Svizzera 19
BE - Belgio 18
TR - Turchia 16
CZ - Repubblica Ceca 14
PH - Filippine 13
PL - Polonia 13
IN - India 10
LB - Libano 10
JP - Giappone 9
CA - Canada 8
EG - Egitto 5
GR - Grecia 5
TH - Thailandia 5
UZ - Uzbekistan 5
IR - Iran 4
TW - Taiwan 4
ES - Italia 3
EU - Europa 3
MD - Moldavia 3
MX - Messico 3
MY - Malesia 3
AU - Australia 2
BG - Bulgaria 2
CO - Colombia 2
HU - Ungheria 2
IL - Israele 2
IQ - Iraq 2
KH - Cambogia 2
PK - Pakistan 2
AF - Afghanistan, Repubblica islamica di 1
CI - Costa d'Avorio 1
DK - Danimarca 1
EC - Ecuador 1
IS - Islanda 1
KG - Kirghizistan 1
LT - Lituania 1
MA - Marocco 1
ME - Montenegro 1
NG - Nigeria 1
NO - Norvegia 1
NZ - Nuova Zelanda 1
PA - Panama 1
PE - Perù 1
PY - Paraguay 1
TN - Tunisia 1
XK - ???statistics.table.value.countryCode.XK??? 1
Totale 5.863
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Città #
Chandler 543
Dublin 431
Singapore 348
Jacksonville 254
Hong Kong 238
San Mateo 228
Boardman 171
Santa Clara 137
Perugia 113
Medford 107
Wilmington 107
Princeton 106
Dong Ket 94
Ann Arbor 89
Altamura 75
Andover 59
Lawrence 49
Beijing 48
Ashburn 47
Bucharest 46
Des Moines 45
Falls Church 33
Dearborn 32
Norwalk 28
Saint Petersburg 25
Vienna 25
Redmond 24
Shanghai 22
Helsinki 20
Brussels 18
Los Angeles 18
Seoul 18
Woodbridge 18
Izmir 11
Redwood City 11
New York 10
Groningen 9
Parma 9
Philadelphia 9
Bologna 8
Dallas 8
Olomouc 8
Cebu City 7
Seodaemun-gu 7
Auburn Hills 6
Collazzone 6
Glasgow 6
Guangzhou 6
Houston 6
Cairo 5
Edinburgh 5
Amsterdam 4
Falkenstein 4
Karaj 4
Milan 4
Moscow 4
Nanjing 4
Renton 4
Simi Valley 4
São Paulo 4
Xiamen 4
Cambridge 3
Chicago 3
Chisinau 3
Delft 3
Den Haag 3
Dumaguete 3
Gwangjin-gu 3
Hachenburg 3
Jinju 3
Kunming 3
Macerata 3
Mumbai 3
Munich 3
Osaka 3
Phoenix 3
Prague 3
Rimini 3
Siena 3
Timisoara 3
Tokyo 3
Villeurbanne 3
Aquila 2
Bangkok 2
Barrie 2
Bengaluru 2
Cannara 2
Chicoutimi 2
Cyberjaya 2
Didcot 2
Dongjak-gu 2
Ealing 2
Frankfurt Am Main 2
Istanbul 2
Jeju City 2
Jinan 2
Kyiv 2
Lausanne 2
Lugano 2
Mainz 2
Totale 3.892
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Nome #
RAGE in the pathophysiology of skeletal muscle. 101
RAGE signalling in myoblasts and embryonal rhabdomyosarcoma cells represses Pax7 expression via p38 MAPK-dependent induction of myogenin. 99
An HMGB1/RAGE/p38 MAPK/Myogenin Axis Modulates Pax7 Expression in Myoblasts by Both Transcriptional and Post-Transcriptional Mechanisms 95
RAGE expression in rhabdomyosarcoma cells results in reduced proliferation, migration, and invasiveness in vitro and tumor growth in vivo 94
Sertoli cells protect C2C12 myotubes against atrophy and induce utrophin expression in canine and human dystrophic myotubes. 89
S100B protein regulates myoblast and macrophage functions in skeletal muscle regeneration 88
Activation of RAGE in myoblasts and rhabdomyosarcoma cells results in downregulation of Pax7 expression 87
Oxidative stress-induced S100B accumulation converts myoblasts into brown adipocytes via an NF-κB/YY1/miR-133 axis and NF-κB/YY1/BMP-7 axis 84
Implication of RAGE and Amphoterin in Myogenesis 80
Amphoterin-induced myogenic differentiation of RAGE-transfected rhabdomyosarcoma TE671 cells 79
Differential expression of S100B protein and RAGE in young and aged human satellite cells. 79
Glyoxalase 1 sustains the metastatic phenotype of prostate cancer cells via EMT control 78
Targeting RAGE as a potential therapeutic approach to Duchenne muscular dystrophy 78
Cellular and molecular mechanisms of sarcopenia: the S100B perspective 77
Toward the identification of receptor for advanced glycation end-products (RAGE) as a muscle biomarker of cancer cachexia. 77
Natural products to counteract muscle atrophy 77
Aged vs young human satellite cells: altered expression of S100B and RAGE, and defective ability in conditioning the medium contribute to impaired myogenic potential. 76
Targeting RAGE prevents muscle wasting and prolongs survival in cancer cachexia 76
Opposing regulatory roles of S100B and amphoterin in myogenic differentiation: RAGE-dependence of amphoterin stimulatory effects vs. RAGE-independence of S100B inhibitory effects 75
Screening of 100 plant extracts for the development of a herbal product effective against muscle atrophy 75
Amphoterin stimulates myogenesis and counteracts the anti-myogenic factors, bFGF and S100B, via RAGE binding 73
RAGE and its ligands, S100B and HMGB1, are molecular determinants of cancer-induced muscle wasting. 72
RAGE signaling in myoblasts and rhabdomyosarcoma cells causes downregulation of Pax7 expression via p38 MAPK activation and upregulation of myogenin expression 71
RAGE-independence of S100B inhibition of myogenic differentiation and myotube formation 71
Sertoli cell-secreted factors have promyogenic and antifibrotic properties on human DMD myoblasts with different mutations. 68
Effects of deletion of RAGE in muscle regeneration: preliminary observations. 67
RAGE engagement in myoblasts modulates proliferation, apoptosis, adhesiveness, migration and invasiveness 66
A braking circuit between pathogen- and danger-sensing signaling pathways restrains lung inflammation: role of S100B protein, RAGE and Toll-like receptors. 66
Genetically-determined hyperfunction of the S100B/RAGE axis is a risk factor for aspergillosis in stem cell transplant recipients 66
The danger signal S100B integrates pathogen- and danger-sensing pathways to restrain inflammation 66
S100B protein differentially regulates myoblast differentiation via direct binding to RAGE and bFGF-mediated activation of FGFR1 in low-density and high-density cultures, respectively 65
Molecular mechanism of S100B-dependent inhibition of myoblast differentiation 65
Employment of Microencapsulated Sertoli Cells as a New Tool to Treat Duchenne Muscular Dystrophy 64
Microencapsulated Sertoli cells sustain myoblast proliferation without affecting the myogenic potential. In vitro data 61
Elucidating the mechanism of S100B-dependent regulation of myoblast differentiation. 60
Levels of S100B protein drive the reparative process in acute muscle injury and muscular dystrophy 59
Functional inactivation of RAGE in myoblasts results in tumor formation 58
Potential role of S100B protein in myogenesis and skeletal muscle regeneration 58
Differential involvement of RAGE and FGFR1 in S100B effects on myoblast differentiation 58
Hypoxia Promotes Danger-mediated Inflammation via Receptor for Advanced Glycation End Products in Cystic Fibrosis 58
The receptor RAGE: a potential molecular target in cancer cachexia 58
Use of Sertoli cells to treat DMD patients is supported by their immunomodulatory rather than immunosuppressive effect 58
S100B causes apoptosis in myoblasts and inhibits myogenic differentiation and myotube formation in a RAGE-independent manner 57
Phosphocaveolin-1 Enforces Tumor Growth and Chemoresistance in Rhabdomyosarcoma 57
Do porcine Sertoli cells represent an opportunity for Duchenne muscular dystrophy? 57
S100B-dependent inhibition of myoblast differentiation: molecular mechanism 56
Young and Aged Human Muscle Satellite Cells Show Differential Expression of S100B Protein and RAGE. 56
Appropriate levels of extracellular S100B protein in injured muscle are required for correct muscle regeneration. 56
Mechanism of S100B-dependent inhibition of myoblast differentiation 55
S100B activates muscle satellite cells via RAGE engagement 55
HMGB1/RAGE modulates Pax7 expression in myoblasts via p38 MAPK-dependent upregulation of myogenin. 55
Effects of RAGE expression in rhabdomyosarcoma cells 54
Complex regulatory effects of extracellular S100B on myoblast differentiation: S100B activates quiescent myoblats and satellite cells 54
Absence of RAGE in an animal experimental model of Duchenne muscular dystrophy results in reduced muscle necrosis and inflammation 54
S100B protein accelerates the activation of quiescent myoblasts and muscle satellite cells. 53
Different intrinsic properties of young and aged human satellite cells. 51
S100B activates quiescent myoblasts and satellite cells 50
Functions of S100 proteins 50
RAGE transduces antiproliferative, pro-apoptotic and anti-tumor signals in myoblasts 49
S100B protein, a damage-associated molecular pattern protein in the brain and heart, and beyond 49
Enforced expression of RAGE in rhabdomyosarcoma cells result in reduced proliferation, migration, and invasiveness in vitro, activation of a myogenic program, and reduced tumor growth in vivo. 49
Novel data support the use of microencapsulated Sertoli cells as a potential treatment of DMD patients. 48
Human muscle satellite cells show age-related differential expression of S100B protein and RAGE. 47
RAGE modulates myoblast proliferation, apoptosis, migration and invasiveness 46
Delayed Muscle Regeneration in RAGE-/- Skeletal Muscles. 46
The many faces of S100B protein: when an extracellular factor inactivates its own receptor and activates another one. 46
S100B engages RAGE or bFGF/FGFR1 in myoblasts depending on its own concentration and myoblast density. Implications for muscle regeneration 46
Defective RAGE activity in embryonal rhabdomyosarcoma cells results in high PAX7 levels that sustain migration and invasiveness 46
Spatiotemporal regulation of Toll-like receptors and RAGE signaling pathways by S100B protein restrains inflammation 45
The novel DMD experimental model, mdx/Ager–/– mouse reveals a role of RAGE in inflammatory processes in dystrophic muscles. 45
S100B inhibits myoblast differentiation via activation of a Ras-MEK-ERK1/2 signaling pathway 45
Receptor for advanced glycation end-products (RAGE) as a biomarker of muscle wasting in cancer conditions 45
Hyperactivated rage in comorbidities as a risk factor for severe covid-19—the role of rage-ras crosstalk 45
S100 proteins in obesity: liaisons dangereuses 44
Causes of elevated serum levels of S100B protein in athletes 43
Involvement of a RAGE/p38MAPK/myogenin axis in cancer cachexia. 43
Re-expression of RAGE in damaged skeletal muscles: RAGE-/- mice show delayed muscle regeneration 42
Differential engagement of RAGE and FGFR1 in muscle satellite cells by S100B protein: involvement of RAGE in satellite cell activation and of FGFR1 on satellite cell expansion. 42
RAGE in tissue homeostasis, repair and regeneration. 42
S100B causes apoptosis in a myoblast cell line in a RAGE-independent manner 41
Reductive stress in striated muscle cells 41
Optimizing therapeutic outcomes of immune checkpoint blockade by a microbial tryptophan metabolite 41
Beneficial effects of horsetail (Equisetum arvense) in experimental models of sarcopenia and osteoporosis 40
RAGE expression in rhabdomyosarcoma cells modulates metastasis formation in vivo. 40
S100B protein regulates myoblast proliferation and differentiation by activating FGFR1 in a bFGF-dependent manner 40
Caveolins and cavins in muscle-derived tumours. 40
S100B's double life: Intracellular regulator and extracellular signal. 39
Identification of Withania somnifera-Silybum marianum-Trigonella foenum-graecum Formulation as a Nutritional Supplement to Contrast Muscle Atrophy and Sarcopenia 39
The amphoterin (HMGB1)/receptor for advanced glycation end products (RAGE) pair modulates myoblast proliferation, apoptosis, adhesiveness, migration, and invasiveness. Functional inactivation of RAGE in L6 myoblasts results in tumor formation in vivo 38
Role of CD45 signaling pathway in Galactoxylomannan-induced T cell damage. 38
HMGB1/RAGE regulates muscle satellite cell homeostasis via p38 MAPK/myogenin dependent repression of Pax7 transcription 38
S100B protein in tissue development, repair and regeneration 38
RAGE expression in rhabdomyosarcoma cells results in myogenic differentiation and reduced proliferation, migration, invasiveness, and tumor growth 37
Regulatory effects of S100B protein on myoblast and muscle satellite cell differentiation depend on cell density and the time of exposure 36
S100B inhibits myogenic differentiation and myotube formation in a RAGE-independent manner 36
S100B protein restrains inflammation via spatiotemporal regulation of pathogen- and danger-sensing signaling pathways. 35
Pharmacological targeting of the receptor for advanced glycation end-products (RAGE) to counteract cancer cachexia 34
RAGE is re-expressed in skeletal muscle satellite cells after muscle injury and deletion of RAGE results in delayed muscle regeneration 34
S100B protein in skeletal muscle regeneration: regulation of myoblast and macrophage functions 34
Equisetum arvense standardized extract hinders age-related sarcopenia 33
Totale 5.687
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Categoria #
all - tutte 25.860
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 25.860
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2019/2020309 0 0 0 0 0 0 92 6 120 39 7 45
2020/2021887 2 69 31 73 284 46 81 6 91 18 75 111
2021/2022938 7 168 26 43 33 20 16 264 19 44 110 188
2022/20231.620 134 273 28 131 133 184 2 76 571 4 60 24
2023/2024737 43 70 29 31 15 18 211 23 80 16 87 114
2024/2025937 55 203 165 106 297 109 2 0 0 0 0 0
Totale 6.105